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Evidence suggests Semaglutide maydecreaseHemoglobin a1c.

14 studies (18 claims)

Moderate consensus

Study Claims

15 of 18
InterventionDirectionEndpointTypePopulationDosageTitle
24 weeks of semaglutide treatmentDecreases - significant improvements inglycated hemoglobin level
Human
patients with non-alcoholic fatty liver disease (NAFLD) or non-alcoholic steatohepatitis (NASH)Not specified in the abstract.Role of semaglutide in the treatment of nonalcoholic fatty liver disease or non-alcoholic steatohepatitis: A systematic review and meta-analysis.cited 30×
high-dose semaglutide (2 mg)Decreases - reveal significant reductions inhemoglobin A1C levels
Human
Glucagon-Like Peptide-1 Agonists: A Practical Overview for Plastic and Reconstructive Surgeons.
once-weekly cagrilintide-semaglutide (2.4 mg each)Decreases - percentage of patients who hadglycated hemoglobin level of 6.5% or less
Human
patientsCagrilintide-Semaglutide in Adults with Overweight or Obesity and Type 2 Diabetes.
subcutaneous semaglutideDecreases - decreasedhemoglobin A1c (HbA1c) level
Human
patients with type 2 diabetesEffect of Oral Semaglutide Compared With Placebo and Subcutaneous Semaglutide on Glycemic Control in Patients With Type 2 Diabetes: A Randomized Clinical Trial.
oral semaglutideDecreases - decreasedhemoglobin A1c (HbA1c) level
Human
patients with type 2 diabetesEffect of Oral Semaglutide Compared With Placebo and Subcutaneous Semaglutide on Glycemic Control in Patients With Type 2 Diabetes: A Randomized Clinical Trial.
oral semaglutideDecreases - reductions were significant vs placebohemoglobin A1c (HbA1c) level
Human
patients with type 2 diabetesEffect of Oral Semaglutide Compared With Placebo and Subcutaneous Semaglutide on Glycemic Control in Patients With Type 2 Diabetes: A Randomized Clinical Trial.
semaglutideDecreases - reducedhemoglobin A1c
Human
adult with CFRD, obesity, and clinical insulin resistanceGlucagon-Like Peptide 1 Agonist Use in an Adult With Cystic Fibrosis-Related Diabetes and Metabolic Syndrome.
semaglutideIncreases - later experiencedhemoglobin A1C (HbA1c) levels
Human
a 36-year-old man with a history of type 2 diabetes mellitus (T2DM) and prior plantar necrotizing fasciitisAcute Duodenal Ulcer Perforation Following Tirzepatide Treatment: A Case Report.
SemaglutideDecreases - significantly reducedhemoglobin A1c (HbA1c) levels
Human
patientsComparative effectiveness of semaglutide
semaglutide at a dose of 1 mgDecreases - estimated mean change from baselineglycated hemoglobin level
Human
patients with type 2 diabetesTirzepatide versus Semaglutide Once Weekly in Patients with Type 2 Diabetes.
once-weekly subcutaneous semaglutide 2.4 mgDecreases - positive changesglycated hemoglobin (HbA1c)
Human
people with obesityOnce-weekly subcutaneous semaglutide 2.4 mg.Cardiometabolic risk factors efficacy of semaglutide in the STEP program.cited 3×
oral semaglutideDecreases - reducedglycated hemoglobin
Human
patients with uncontrolled T2D on various background therapiesNot applicable.Integrating oral semaglutide into clinical practice in primary care: for whom, when, and how?cited 16×
injectable semaglutide or tirzepatideDecreases - mean (SD) absolute reduction in percent glycated hemoglobin at 1 year wasglycated hemoglobin
Human
adults with overweight or obesity without type 2 diabetes with early discontinuationChanges in weight and glycemic control following obesity treatment with semaglutide or tirzepatide by discontinuation status.
injectable semaglutide or tirzepatideDecreases - mean (SD) absolute reduction in percent glycated hemoglobin at 1 year wasglycated hemoglobin
Human
adults with overweight or obesity without type 2 diabetes with late discontinuationChanges in weight and glycemic control following obesity treatment with semaglutide or tirzepatide by discontinuation status.
injectable semaglutide or tirzepatideDecreases - mean (SD) absolute reduction in percent glycated hemoglobin at 1 year wasglycated hemoglobin
Human
adults with overweight or obesity without type 2 diabetes with non-discontinuationChanges in weight and glycemic control following obesity treatment with semaglutide or tirzepatide by discontinuation status.