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Modulation of circulating levels of advanced glycation end products and its impact on intima-media thickness of both common carotid arteries: CORDIOPREV randomised controlled trial.

Cardiovascular diabetology
October 14, 2024
Francisco M Gutierrez-Mariscal et al. (12 authors)
Journal ArticleRandomized Controlled TrialComparative StudyResearch Support, Non-U.S. Gov'tHuman StudyClinical
Study Details

Study Goal

The researchers aimed to determine whether the Mediterranean diet's reduction in atherosclerosis progression is associated with modulation of circulating AGE levels in coronary heart disease (CHD) patients.

Results Summary

The Mediterranean diet improved AGE metabolism (increased AGER1 and GloxI gene expression, GSH, and sRAGE levels) and better managed circulating AGE levels compared to a low-fat diet, potentially reducing atherosclerosis progression in CHD patients.

Population

1002 patients with coronary heart disease (CHD).

Effective Dosage

Not specified

Duration

5 years

Interactions

None mentioned

Extracted Claims (11)
InterventionDirectionEndpointPopulationDosageImpactClaim #
Mediterranean diet
no change
MG levels
patients with coronary heart disease (CHD)
-
did not affect
#1
low-fat diet
increase
MG levels
patients with coronary heart disease (CHD)
-
significantly increased
#2
Mediterranean diet
decrease
MG levels
patients with coronary heart disease (CHD)
-
leading to lower
#3
Mediterranean diet
increase
AGE metabolism
Non-increased IMT-CC patients
-
produced an upregulation
#4
Mediterranean diet
increase
AGER1 gene expression
Non-increased IMT-CC patients
-
increased
#5
Mediterranean diet
increase
GloxI gene expression
Non-increased IMT-CC patients
-
increased
#6
Mediterranean diet
increase
GSH levels
Non-increased IMT-CC patients
-
increased
#7
Mediterranean diet
increase
sRAGE levels
Non-increased IMT-CC patients
-
increased
#8
Mediterranean diet
increase
MG levels
Increased IMT-CC patients
-
increased
#9
Mediterranean diet
increase
MG levels
Increased IMT-CC patients
-
this increment was lower
#10
Mediterranean diet
decrease
atherosclerosis
patients with CHD
-
reduces the progression
#11
Abstract

BACKGROUND: Increasing evidence supports the role of advanced glycation end products (AGEs) in atherosclerosis in both diabetic and non-diabetic patients, suggesting that therapeutic strategies targeting AGEs may offer potential benefits in this population. The Mediterranean diet is associated with improved biomarkers and anthropometric measurements related with atherosclerosis in addition to its ability to modulate AGE metabolism. Our aim was to determine whether the reduction in atherosclerosis progression (measured by changes in intima-media thickness of both common carotid arteries (IMT-CC)), observed after consumption of a Mediterranean diet compared to a low-fat diet, is associated with a modulation of circulating AGE levels in patients with coronary heart disease (CHD). METHODS: 1002 CHD patients were divided in: (1) Non-increased IMT-CC patients, whose IMT-CC was reduced or not changed after dietary intervention and (2) Increased IMT-CC patients, whose IMT-CC was increased after dietary intervention. Serum AGE levels (methylglyoxal-MG and Nε-Carboxymethyllysine-CML) and parameters related to AGE metabolism (AGER1 and GloxI mRNA and sRAGE levels) and reduced glutathione (GSH) levels were measured before and after 5-years of dietary intervention. RESULTS: The Mediterranean diet did not affect MG levels, whereas the low-fat diet significantly increased them compared to baseline (p = 0.029), leading to lower MG levels following the Mediterranean diet than the low-fat diet (p < 0.001). The Mediterranean diet, but not the low-fat diet, produced an upregulation of AGE metabolism, with increased AGER1 and GloxI gene expression as well as increased GSH and sRAGE levels in Non-increased IMT-CC patients (all p < 0.05). Although the Mediterranean diet increased MG levels in Increased IMT-CC patients, this increment was lower compared to the low-fat diet (all p < 0.05). CONCLUSIONS: Our results suggest that an improvement in modulation of AGE metabolism, which facilitates better management of circulating AGE levels, may be one of the mechanisms through which the Mediterranean diet, compared to a low-fat diet, reduces the progression of atherosclerosis in patients with CHD. Trial registration https://clinicaltrials.gov/ct2/show/NCT00924937 , Clinicaltrials.gov number, NCT00924937.

Medical Subject Headings (MeSH)
HumansGlycation End Products, AdvancedMaleCarotid Intima-Media ThicknessDiet, MediterraneanFemaleMiddle AgedAgedReceptor for Advanced Glycation End ProductsBiomarkersCarotid Artery, CommonTreatment OutcomeDiet, Fat-RestrictedCarotid Artery DiseasesTime FactorsDisease ProgressionAntigens, NeoplasmLactoylglutathione LyaseMitogen-Activated Protein Kinases
Study Links
Quality Scores
SafetyNot Assessed
Efficacy85/10
Quality90/10
Citation Metrics
Total Citations3
Citations/Year3.0
Research Impact Scores
APT Score0.25
Weight Score2.98
Normalized Score0.72