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Omega-3 fatty acids prevent nicotine withdrawal-induced impairment of learning and memory via affecting oxidative status, inflammatory response, cholinergic activity, BDNF and amyloid-B in rat hippocampal tissues.

Life sciences
November 1, 2023
S Mohammad Ahmadi-Soleimani et al. (5 authors)
Journal ArticleAnimal Study
Study Details

Study Goal

The researchers aimed to determine whether Omega-3 fatty acids could prevent the adverse effects of adolescent nicotine withdrawal on spatial and avoidance memory in male rats.

Results Summary

Omega-3 fatty acids significantly prevented nicotine withdrawal-induced adverse effects by restoring biochemical disturbances, including oxidative stress, inflammation, and neurotrophic capacity in hippocampal tissues. The study suggests Omega-3 fatty acids as a safe and effective therapeutic strategy for memory dysfunction associated with nicotine abstinence.

Population

Male Wistar rats (adolescent)

Effective Dosage

Not specified

Duration

Not specified

Interactions

None mentioned

Extracted Claims (7)
InterventionDirectionEndpointPopulationDosageImpactClaim #
adolescent nicotine exposure followed by a period of drug cessation
increase
behavioral indices of learning and memory
male Wistar rats
-
exacerbates
#1
adolescent nicotine exposure followed by a period of drug cessation
increase
oxidative and inflammatory markers
male Wistar rats
-
lead to elevation of
#2
adolescent nicotine exposure followed by a period of drug cessation
decrease
neurotrophic capacity
male Wistar rats
-
reduction of
#3
adolescent nicotine exposure followed by a period of drug cessation
increase
AChE activity
male Wistar rats
-
increased
#4
co-administration of nicotine with Omega-3 fatty acids
decrease
nicotine withdrawal-induced adverse effects
male Wistar rats
-
significantly prevents
#5
co-administration of nicotine with Omega-3 fatty acids
neutral
the mentioned biochemical disturbances
male Wistar rats
-
restoration of
#6
Omega-3 fatty acids
decrease
the adverse effects of adolescent nicotine withdrawal on spatial and avoidance memory
male rats
-
prevent
#7
Abstract

In the present study, the main objective was to reveal whether treatment by Omega-3 fatty acids could prevent the adverse effects of adolescent nicotine withdrawal on spatial and avoidance memory in male rats. For this purpose, Morris water maze and passive avoidance tests were performed on male Wistar rats and the hippocampal levels of oxidative stress markers, inflammatory indices, brain-derived neurotrophic factor, nitrite, amyloid-B and acetylcholinesterase (AChE) were measured. Moreover, density of dark neurons were assessed in CA1 and CA3 regions. Results showed that adolescent nicotine exposure followed by a period of drug cessation exacerbates the behavioral indices of learning and memory through affecting a variety of biochemical markers within the hippocampal tissues. These changes lead to elevation of oxidative and inflammatory markers, reduction of neurotrophic capacity and increased AChE activity in hippocampal tissues. In addition, it was observed that co-administration of nicotine with Omega-3 fatty acids significantly prevents nicotine withdrawal-induced adverse effects through restoration of the mentioned biochemical disturbances. Therefore, we suggest administration of Omega-3 fatty acids as a safe, inexpensive and effective therapeutic strategy for prevention of memory dysfunctions associated with nicotine abstinence during adolescence.

Medical Subject Headings (MeSH)
RatsMaleAnimalsNicotineRats, WistarAcetylcholinesteraseBrain-Derived Neurotrophic FactorHippocampusSubstance Withdrawal SyndromeOxidative StressAmyloidCholinergic AgentsFatty Acids, Omega-3Maze LearningMemory Disorders
Study Links
Quality Scores
Safety85
Efficacy90/10
Quality80/10
Citation Metrics
Total Citations9
Citations/Year4.5
Relative Citation Ratio2.33
NIH Percentile78.9%
Research Impact Scores
APT Score0.25
Weight Score2.10
Normalized Score0.86
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