Panacea Index Logo

Command Palette

Search for a command to run...

Ketogenic Diet Suppressed T-Regulatory Cells and Promoted Cardiac Fibrosis via Reducing Mitochondria-Associated Membranes and Inhibiting Mitochondrial Function.

Oxidative medicine and cellular longevity
January 1, 2021
Jun Tao et al. (12 authors)
Journal ArticleHuman StudyAnimal Study
Extracted Claims (11)
InterventionDirectionEndpointPopulationDosageImpactClaim #
Ketogenic diet (KD)
neutral
cardiac function
diabetic cardiomyopathy (DCM)
-
effects and the underlined mechanisms
#1
Ketogenic diet (KD)
neutral
cardiac safety
diabetic patients
-
effects and the underlined mechanisms
#2
Ketogenic diet (KD)
neutral
cardiac efficiency
diabetic patients
-
effects and the underlined mechanisms
#3
different diets (regular or KD)
neutral
cardiac function
db/db mice
-
determined
#4
different diets (regular or KD)
neutral
interstitial fibrosis
db/db mice
-
determined
#5
different diets (regular or KD)
neutral
T-regulatory cell (Treg) number
db/db mice
-
evaluated
#6
different diets (regular or KD)
neutral
T-regulatory cell (Treg) functions
db/db mice
-
evaluated
#7
ketone body (KB)
neutral
fatty acid (FA) metabolism
-
-
assessed
#8
ketone body (KB)
neutral
glucose metabolism
-
-
assessed
#9
ketone body (KB)
neutral
mitochondria-associated endoplasmic reticulum membranes (MAMs)
-
-
assessed
#10
ketone body (KB)
neutral
mitochondrial respiration
-
-
assessed
#11
Abstract

Ketogenic diet (KD) is popular in diabetic patients but its cardiac safety and efficiency on the heart are unknown. The aim of the present study is to determine the effects and the underlined mechanisms of KD on cardiac function in diabetic cardiomyopathy (DCM). We used db/db mice to model DCM, and different diets (regular or KD) were used. Cardiac function and interstitial fibrosis were determined. T-regulatory cell (Treg) number and functions were evaluated. The effects of ketone body (KB) on fatty acid (FA) and glucose metabolism, mitochondria-associated endoplasmic reticulum membranes (MAMs), and mitochondrial respiration were assessed. The mechanisms

Medical Subject Headings (MeSH)
AnimalsCardiovascular DiseasesDiet, KetogenicFibrosisHumansMaleMiceMitochondriaT-Lymphocytes, Regulatory
Study Links
PubMed ID33959215
Related Supplements