Panacea Index Logo

Command Palette

Search for a command to run...

Role of melatonin in periodontal disease - A systematic review.

Indian journal of dental research : official publication of Indian Society for Dental Research
January 1, 2020
S Swarna Meenakshi et al. (2 authors)
Journal ArticleSystematic ReviewHuman Study
Study Details

Study Goal

The researchers aimed to systematically evaluate the role of melatonin in periodontal disease, focusing on its immunomodulatory, antioxidant, and angiogenic properties.

Results Summary

Melatonin showed beneficial effects in inflammatory oral pathologies, particularly periodontal diseases, by inhibiting bone resorption, destroying reactive oxygen species, and stimulating osteoblastic differentiation. Lower melatonin levels in patients with chronic periodontitis suggest its protective role against oxidative stress, but optimal dosage and application methods remain unclear.

Population

Patients with chronic periodontitis (as indicated by melatonin levels in GCF, saliva, and serum).

Effective Dosage

Not specified

Duration

Not specified

Interactions

None mentioned

Extracted Claims (12)
InterventionDirectionEndpointPopulationDosageImpactClaim #
melatonin
neutral
immune function
-
-
has immunomodulatory properties
#1
melatonin
neutral
oxidative stress
-
-
has antioxidant properties
#2
melatonin
decrease
inflammation
-
-
protects against inflammation
#3
melatonin
decrease
cellular damage
-
-
protects against cellular damage
#4
melatonin
increase
angiogenesis
-
-
has potent angiogenic function
#5
melatonin
neutral
inflammatory oral pathologies
-
-
may have beneficial effects
#6
melatonin
decrease
bone resorption
patients with periodontal diseases
-
inhibit bone resorption
#7
melatonin
decrease
reactive oxygen species
patients with periodontal diseases
-
destroy reactive oxygen species
#8
melatonin
increase
osteoblastic differentiation
patients with periodontal diseases
-
stimulates osteoblastic differentiation
#9
salivary melatonin
neutral
periodontal diseases
-
-
could act as a risk indicator
#10
melatonin
decrease
melatonin levels in GCF, Saliva, Serum
patients suffering from chronic periodontitis
-
levels are lowered
#11
melatonin
decrease
tissue damage caused by oxidative stress
-
-
may play a pivotal role in protecting the tissue
#12
Abstract

BACKGROUND AND AIMS: Melatonin is an indolamine that is primarily secreted by the pineal gland. It has immunomodulatory as well as antioxidant properties. It is a potent anti-oxidant that protects against inflammation and cellular damage caused by reactive oxygen species, also has potent angiogenic function that adds on to the benefits of melatonin. As a result of these actions, melatonin may be useful as an adjuvant in the treatment of various conditions in the oral cavity. The aim of this study is to systematically evaluate the role of melatonin in periodontal disease. METHODS: An extensive review of the scientific literature was carried out using PubMed, Science Direct, Google Scholar and the Cochrane base. Research articles were collected upto December 2017. RESULTS: Melatonin may have beneficial effects in certain inflammatory oral pathologies, mainly periodontal diseases where they inhibit bone resorption destroy reactive oxygen species, stimulates osteoblastic differentiation. Salivary melatonin could also act as a risk indicator for periodontal diseases. CONCLUSION: Many studies showed that the melatonin levels in GCF, Saliva, Serum of patients suffering from chronic periodontitis is lowered suggesting that may play a pivotal role in protecting the tissue from damage caused by oxidative stress. However, there exists no data on the concentration needed, method of application for potential benefits. Randomized clinical trials in this field are needed to fill the lacunae and better improve our understanding.

Medical Subject Headings (MeSH)
AntioxidantsHumansMelatoninOxidative StressPeriodontal DiseasesSaliva
Study Links
Quality Scores
SafetyNot Assessed
Efficacy70/10
Quality60/10
Citation Metrics
Total Citations12
Citations/Year2.4
Relative Citation Ratio1.18
NIH Percentile56.3%
Research Impact Scores
APT Score0.75
Weight Score1.98
Normalized Score0.60
Related Supplements