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Interaction of Melatonin and BMP-6 in Ovarian Steroidogenesis.

Vitamins and hormones
January 1, 2018
Fumio Otsuka
Journal ArticleResearch Support, Non-U.S. Gov'tReviewHuman StudyMolecular Study
Study Details

Study Goal

The researchers aimed to determine whether melatonin could counteract the inhibitory effects of BMP-6 on progesterone production in granulosa cells, potentially preserving ovarian function.

Results Summary

Melatonin treatment impaired BMP-6's inhibition of FSH-induced progesterone production and suppressed BMP-6-induced Smad signaling in granulosa cells, suggesting a regulatory role in BMP receptor signaling. This indicates melatonin may help maintain progesterone production by suppressing BMP-6 signaling, particularly relevant in conditions like polycystic ovary syndrome.

Population

Granulosa cells (in vitro study)

Effective Dosage

Not specified

Duration

Not specified

Interactions

None mentioned

Extracted Claims (9)
InterventionDirectionEndpointPopulationDosageImpactClaim #
BMP-6
decrease
follicle-stimulating hormone (FSH) actions
-
-
exert an inhibitory effect
#1
BMP-6
decrease
adenylate cyclase activity downstream of the FSH receptor
-
-
suppressing
#2
melatonin treatment
decrease
inhibition of FSH-induced progesterone production by BMP-6
granulosa cells
-
impaired
#3
melatonin
decrease
intracellular Smad signaling induced by BMP-6
granulosa cells
-
suppressed
#4
melatonin
neutral
BMP receptor signaling
granulosa cells
-
has a regulatory role
#5
-
increase
expression of BMP-6 in granulosa cells
patients with polycystic ovary syndrome
-
increased
#6
melatonin
increase
progesterone production
-
-
play an important role in the maintenance
#7
melatonin
decrease
BMP-6 signaling
-
-
suppressing
#8
melatonin
increase
ovarian function
-
-
leading to the preservation
#9
Abstract

The bone morphogenetic protein (BMP) system in the ovary plays a physiological role as a luteinization inhibitor in growing follicles. BMP-6 secreted from oocytes and granulosa cells can exert an inhibitory effect on follicle-stimulating hormone (FSH) actions by suppressing adenylate cyclase activity downstream of the FSH receptor. The inhibition of FSH-induced progesterone production by BMP-6 is impaired by melatonin treatment in granulosa cells. Intracellular Smad signaling induced by BMP-6 is suppressed by melatonin, suggesting that melatonin has a regulatory role in BMP receptor signaling in granulosa cells. Since the expression of BMP-6 in granulosa cells is increased in patients with polycystic ovary syndrome, melatonin may play an important role in the maintenance of progesterone production by suppressing BMP-6 signaling, leading to the preservation of ovarian function.

Medical Subject Headings (MeSH)
AnimalsBone Morphogenetic Protein 6EstrogensEstrous CycleFemaleFollicular PhaseGranulosa CellsHumansHypothalamo-Hypophyseal SystemMelatoninMenstrual CycleModels, BiologicalOocytesOogenesisOvaryReceptors, MelatoninSignal TransductionSteroidsTheca Cells
Study Links
Quality Scores
SafetyNot Assessed
Efficacy75/10
Quality65/10
Citation Metrics
Total Citations17
Citations/Year2.4
Relative Citation Ratio0.99
NIH Percentile49.9%
Research Impact Scores
APT Score0.05
Weight Score0.93
Normalized Score0.63
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