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Effects of semaglutide on beta cell function and glycaemic control in participants with type 2 diabetes: a randomised, double-blind, placebo-controlled trial.

Diabetologia
August 1, 2017
Christoph Kapitza et al. (5 authors)
Journal ArticleRandomized Controlled TrialResearch Support, Non-U.S. Gov'tHuman Study
Extracted Claims (9)
InterventionDirectionEndpointPopulationDosageImpactClaim #
semaglutide
increase
first-phase insulin secretion
participants with type 2 diabetes
2.5-fold
significantly increased
#1
semaglutide
increase
second-phase insulin secretion
participants with type 2 diabetes
2.5-fold
significantly increased
#2
semaglutide
increase
insulin AUC
participants with type 2 diabetes
2.5-fold
significantly increased
#3
semaglutide
increase
C-peptide AUC
participants with type 2 diabetes
2.5-fold
significantly increased
#4
semaglutide
decrease
glucose AUC
participants with type 2 diabetes
50%
significantly reduced
#5
semaglutide
decrease
fasting plasma glucose
participants with type 2 diabetes
2.5 mmol/L
significantly reduced
#6
semaglutide
decrease
HbA1c
participants with type 2 diabetes
1.2%
significantly reduced
#7
semaglutide
increase
beta cell function
participants with type 2 diabetes
-
significantly improved
#8
semaglutide
increase
glycaemic control
participants with type 2 diabetes
-
significantly improved
#9
Abstract

AIMS/HYPOTHESIS: Semaglutide is a glucagon-like peptide-1 analogue in development for the treatment of type 2 diabetes. Its effects on first- and second-phase insulin secretion and other measures of beta cell function and glycaemic control were assessed. METHODS: In this single-centre, double-blind, placebo-controlled, parallel-group trial, conducted at the Profil Institut für Stoffwechselforschung, Germany, 75 adult (aged 18-64 years) participants with type 2 diabetes (eligibility: HbA RESULTS: In total, 37 participants received semaglutide and 38 received placebo. Following IVGTT, for insulin, both AUC CONCLUSIONS/INTERPRETATION: Twelve weeks of once-weekly treatment with semaglutide significantly improved beta cell function and glycaemic control in participants with type 2 diabetes. TRIAL REGISTRATION: ClinicalTrials.gov NCT02212067 FUNDING: The study was funded by Novo Nordisk A/S.

Medical Subject Headings (MeSH)
AdolescentAdultBlood GlucoseDiabetes Mellitus, Type 2Double-Blind MethodFemaleGlucagon-Like PeptidesGlycated HemoglobinHumansHypoglycemic AgentsInsulin-Secreting CellsMetforminMiddle AgedPregnancyYoung Adult
Study Links
PubMed ID28526920
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