Panacea Index Logo

Command Palette

Search for a command to run...

CYP2E1 and oxidant stress in alcoholic and non-alcoholic fatty liver disease.

Journal of hepatology
February 1, 2013
Tung-Ming Leung et al. (2 authors)
Journal ArticleResearch Support, N.I.H., ExtramuralReviewHuman Study
Study Details

Study Goal

The researchers aimed to summarize the role of CYP2E1 in alcoholic and non-alcoholic fatty liver diseases, particularly focusing on its contribution to ROS generation and disease progression.

Results Summary

The abstract highlights that high-fat diet contributes to fatty liver diseases by inducing oxidant stress and ROS generation, with CYP2E1 playing a critical role in this process. It also notes that alcohol further induces CYP2E1, exacerbating liver injury.

Population

Not specified (general discussion of ALD and NAFLD mechanisms)

Effective Dosage

Not available

Duration

Not specified

Interactions

None mentioned

Extracted Claims (8)
InterventionDirectionEndpointPopulationDosageImpactClaim #
alcohol consumption
increase
hepatocellular injury and inflammation
patients with ALD
-
leading to
#1
high fat diet
increase
hepatocellular injury and inflammation
patients with NAFLD
-
leading to
#2
obesity
increase
hepatocellular injury and inflammation
patients with NAFLD
-
leading to
#3
diabetes
increase
hepatocellular injury and inflammation
patients with NAFLD
-
leading to
#4
oxidant stress
increase
pathogenesis
patients with ALD and NAFLD
-
is a major contributing factor to
#5
generation of reactive oxygen species (ROS)
increase
progression of fatty liver to steatohepatitis
-
-
is key for
#6
Cytochrome P450 2E1 (CYP2E1)
increase
ROS generation
-
-
plays a critical role in
#7
alcohol
increase
CYP2E1
-
-
induced by
#8
Abstract

Alcoholic (ALD) and non-alcoholic fatty liver diseases (NAFLD) are clinical conditions leading to hepatocellular injury and inflammation resulting from alcohol consumption, high fat diet, obesity and diabetes, among others. Oxidant stress is a major contributing factor to the pathogenesis of ALD and NAFLD. Multiple studies have shown that generation of reactive oxygen species (ROS) is key for the progression of fatty liver to steatohepatitis. Cytochrome P450 2E1 (CYP2E1) plays a critical role in ROS generation and CYP2E1 is also induced by alcohol itself. This review summarizes the role of CYP2E1 in ALD and NAFLD.

Medical Subject Headings (MeSH)
Cytochrome P-450 CYP2E1Disease ProgressionFatty LiverHumansInsulin ResistanceLiverLiver Diseases, AlcoholicNon-alcoholic Fatty Liver DiseaseOxidative StressReactive Oxygen Species
Study Links
Quality Scores
SafetyNot Assessed
Quality70/10
Citation Metrics
Total Citations371
Citations/Year30.9
Relative Citation Ratio12.75
NIH Percentile98.6%
Research Impact Scores
APT Score0.75
Weight Score1.72
Normalized Score0.54
Related Supplements
CYP2E1 and oxidant stress in alcoholic and non-alcoholic fat... | Panacea Index