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Melatonin promotes embryonic development and reduces reactive oxygen species in vitrified mouse 2-cell embryos.

Journal of pineal research
April 1, 2012
Chao Gao et al. (8 authors)
Journal ArticleResearch Support, Non-U.S. Gov'tAnimal StudyMolecular Study
Extracted Claims (7)
InterventionDirectionEndpointPopulationDosageImpactClaim #
vitrification by the open-pulled straw (OPS) method
increase
reactive oxygen species (ROS) formation
Two-cell embryos of mouse
-
caused
#1
vitrification by the open-pulled straw (OPS) method
decrease
development of the embryos
Two-cell embryos of mouse
-
jeopardized
#2
melatonin at different concentrations (10(-3), 10(-5), 10(-7), 10(-9), 10(-11) m)
decrease
ROS production
vitrified embryos
-
significantly suppresses
#3
melatonin at different concentrations (10(-3), 10(-5), 10(-7), 10(-9), 10(-11) m)
increase
embryonic development
vitrified embryos
-
promotes
#4
melatonin
neutral
beneficial effects on vitrified 2-cell embryos of mouse
vitrified 2-cell embryos of mouse
-
were
#5
melatonin
no change
melatonin receptor (MT1 and MT2) independent
vitrified 2-cell embryos of mouse
-
were
#6
melatonin
neutral
protective effects on vitrified embryonic development
vitrified embryos
-
may account for
#7
Abstract

Two-cell embryos of mouse were vitrified by the open-pulled straw (OPS) method. The vitrified embryos were warmed and introduced into M16 medium for culture that contains melatonin at different concentrations (10(-3), 10(-5), 10(-7), 10(-9), 10(-11) m). This process caused reactive oxygen species (ROS) formation and jeopardized the development of the embryos. Melatonin, at different concentrations, significantly suppresses ROS production and promotes embryonic development in vitrified embryos compared with untreated ones. The mechanistic studies indicated that the beneficial effects of melatonin on vitrified 2-cell embryos of mouse were melatonin receptor (MT1 and MT2) independent. The direct free radical scavenging activity, the enhancement of endogenous glutathione levels, and the anti-apoptotic capacity of melatonin may account for its protective effects on vitrified embryonic development.

Medical Subject Headings (MeSH)
AnimalsBase SequenceDNA PrimersEmbryo, MammalianEmbryonic DevelopmentMelatoninMiceReactive Oxygen Species
Study Links
PubMed ID22225541
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