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cJun NH2-terminal kinase 1 (JNK1): roles in metabolic regulation of insulin resistance.

Trends in biochemical sciences
September 1, 2010
Guadalupe Sabio et al. (2 authors)
Journal ArticleResearch Support, N.I.H., ExtramuralResearch Support, Non-U.S. Gov'tReviewAnimal Study
Study Details

Study Goal

The researchers aimed to investigate the role of JNK1 in high-fat diet-induced obesity and insulin resistance in mice.

Results Summary

The study found that a high-fat diet activated the JNK1 pathway, leading to insulin resistance and obesity in mice, while JNK1 ablation prevented these effects. JNK1's role in insulin resistance was separable from its effects on obesity, suggesting it as a potential drug target for metabolic disorders.

Population

Mice (germ-line Jnk1-ablated and wild-type)

Effective Dosage

Not specified

Duration

Not specified

Interactions

None mentioned

Extracted Claims (6)
InterventionDirectionEndpointPopulationDosageImpactClaim #
Feeding a high-fat diet
increase
the JNK1 signaling pathway
mice
-
causes activation
#1
Feeding a high-fat diet
increase
insulin resistance
mice
-
causes
#2
Feeding a high-fat diet
increase
obesity
mice
-
causes
#3
Germ-line ablation of Jnk1
decrease
diet-induced obesity
mice
-
prevents
#4
Germ-line ablation of Jnk1
decrease
insulin resistance
mice
-
prevents
#5
JNK1
neutral
insulin resistance
-
-
plays multiple roles in the regulation
#6
Abstract

The cJun NH(2)-terminal kinase isoform JNK1 is implicated in the mechanism of obesity-induced insulin resistance. Feeding a high-fat diet causes activation of the JNK1 signaling pathway, insulin resistance, and obesity in mice. Germ-line ablation of Jnk1 prevents both diet-induced obesity and insulin resistance. Genetic analysis indicates that the effects of JNK1 on insulin resistance can be separated from effects of JNK1 on obesity. Emerging research indicates that JNK1 plays multiple roles in the regulation of insulin resistance, including altered gene expression, hormone/cytokine production, and lipid metabolism. Together, these studies establish JNK1 as a potential pharmacological target for the development of drugs that might be useful for the treatment of insulin resistance, metabolic syndrome, and type 2 diabetes.

Medical Subject Headings (MeSH)
AnimalsDietHumansInsulin ResistanceInsulin-Secreting CellsMiceMitogen-Activated Protein Kinase 8Obesity
Study Links
Quality Scores
SafetyNot Assessed
Efficacy75/10
Quality85/10
Citation Metrics
Total Citations133
Citations/Year8.9
Relative Citation Ratio2.97
NIH Percentile84.8%
Research Impact Scores
APT Score0.25
Weight Score0.74
Normalized Score0.67
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